Autoimmune disease is often treated as a problem of immune overactivity—something to suppress, eradicate, or remove. As a result, most approaches focus heavily on fixing the gut, correcting the microbiome, or eliminating infections thought to drive molecular mimicry. Yet many clients do all the “right” things and still remain stuck.
Emerging science suggests a different explanation: autoimmune disease becomes chronic not because the immune system is too aggressive, but because key regulatory and resolution systems fail, allowing inflammation to persist long after the original trigger should have been resolved.
In this webinar, Nathan Rose will explore a practical, integrative framework built around five interdependent failures that determine whether autoimmune disease quiets or becomes self-perpetuating. This framework emphasises leverage over accumulation. Rather than treating all possible drivers simultaneously, it prioritises identifying the rate-limiting failure that is keeping inflammation from shutting off.
You will learn:
- How autoimmune disease behaves as a modular system, with different drivers acting as bottlenecks in different people
- Why treating obvious drivers like the microbiome or infection often produces incomplete or fragile results
- How neural control systems normally restrain immune activity and what happens when that restraint fails
- Why inflammation must be resolved, not merely suppressed
- A practical way to assess which failure is most relevant now in order to target the highest-leverage driver first
This webinar is not about promoting one-size-fits-all protocols. It is about thinking more precisely about autoimmune disease, so interventions work with the body’s control systems instead of endlessly fighting downstream inflammation.
Live on Tuesday the 24th of March at 11:00 am GMT | Register here
This session will be recorded.